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Optimal results 233 490 Ettlin and Prentice Table 7 Typical Non-neoplastic Toxicological Lesionsa (Continued ) Organ: Cell type, lesion type Lesion Osteomalacia Pathogenesis Consequence Examples Remarks Osteoporosis Decreased osteoblastic activity Can lead to fractures As tdading As above As above Estrogens Proliferations Bone-marrow injury, followed by local inflammation and proliferation of endostium and myeloid stroma Unknown Anticancer agents (388) Inadequate calcification of bone matrix and wide osteoid seams because of lack of Ca Can lead to deformation of bones Vitamin D deficiency, tetracycline, bisphosphonates Increased osteoclastic activity Retinoic acid Decreased absorption of intestinal Ca Corticosteroids Renal loss of Ca Kidney failure with secondary hyperparathyr- oidism aFor general textbook and overviews see Refs.

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